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GDMT for HFrEF typically includes evidence-based beta-blockers, angiotensin receptor neprilysin inhibitors (ARNIs), angiotensin receptor blockers (ARBs), angiotensin-converting enzyme (ACE) inhibitors, mineralocorticoid receptor antagonists (MRA), and sodium-glucose cotransporter 2 inhibitors (SGLT2is), particularly in patients with obesity or T2DM [53]

However, beta cells lack cytosolic phosphoenolpyruvate carboxykinase and fructose-1,6-bisphosphatase, indicating that PC does not serve a gluconeogenic role in these cells

Once destroyed, the cells can no longer produce insulin [1]

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Meanwhile, the GH-releasing hormone (GHRH), secreted from the hypothalamus, was known to stimulate GH secretion from the pituitary gland