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doi: 10.1093/ecco-jcc/jjaf074 80 DaneseSNeurathMFKopoAZakkoSFSimmonsTCFogelRet al
The hypothesis driving its design was straightforward but unverified at the time: that engaging GIP and GLP-1 receptors simultaneously, pathways which are normally co-secreted from the gut following a meal, would produce additive or synergistic effects on insulin secretion, gastric motility, and downstream metabolic markers beyond what either pathway could deliver alone
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