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Whether PBAT-MPs-induced SCFA depletion drives microglial lipotoxicity through the mTORC1ISR axis remains unknown

Its prominent mechanism lies in the dual mode of scaffold degradation to clear HO + sustained drug release for anti-inflammatory effects: the scaffold effectively scavenges ROS to alleviate oxidative stress, while drug release inhibits inflammation and blocks apoptosis [68]

Targeting the ERAD pathway via inhibition of signal peptide peptidase for antiparasitic therapeutic design

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Leite et al., 2013