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The experimental design for determining the oxygen consumption rate of IDYs and other antioxidants was an adaptation of those previously described by Navarro et al

By binding to these metabolic waste products and exporting them out of the mitochondria, ALCAR frees up Coenzyme A, ensuring that the Krebs cycle can continue churning out the ATP required for everything from muscle contraction to complex cognitive processing

It is acknowledged that T2DM patients with metabolic syndrome had significant features of oxidative stress (64, 65), which may associate with faster NPDR progression