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10.1039/d0fo02274h Food Funct
in fact, these treatments promoted UPR mt and mitophagy, restoring mitochondrial and organismal function, and even stimulated the reduction of amyloid deposits in these models 114
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It binds to NAPQI, preventing the compound from harming liver tissue
This manuscript is a review article and does not involve human participants or animals
The results showed that there was a dose-dependent migration of HUVECs toward CCL11, indicating that CCL11 released by THP-1 cells can affect endothelial cell function by its chemotaxin essence (Fig
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