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If side effects persist or weight loss has stalled, the split may not be providing the benefits you hoped for
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In: Ferri FF, ed
Functioning independently of its canonical unfolded protein response role, the endoplasmic reticulum-resident protein IRE1 governs ferroptotic susceptibility through an evolutionarily conserved mechanism wherein its endoribonuclease activity degrades transcripts encoding glutathione biosynthesis factors GCLC and SLC7A11, rendering IRE1-deficient cells resistant while overexpressing cells hyper-sensitive to this death pathway (55)
10.1021/acs.jmedchem.6b01031 67 KramrE
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