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The 2020 systematic review by Galeano and colleagues across 34 eligible studies confirmed the mechanism: PDRN promotes physiological tissue repair through salvage-pathway nucleotide recycling and A2A receptor activation [32]

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The copper in GHK-Cu is chelated (bound to the peptide), which further limits its bioavailability for systemic absorption

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Hence, FOSL1 and HSPA1A can potentially serve as useful inflammatory markers for copper compounds