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The detailed results of that late-stage study were not published as a full peer-reviewed clinical paper, which limits independent appraisal
AOD 9604, by contrast, is studied for direct effects at the adipocyte level in research preclinical models independent of the growth hormone receptor and, based on research published in Endocrinology , independent of beta-3 adrenergic signaling as well [6]
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Research suggests it may work through different mechanisms, potentially involving direct interaction with adipocyte membranes or intracellular signaling pathways distinct from classical GH receptor activation
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