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Intraperitoneal exendin-4 was shown to interrupt and even revert the progression of drug-induced nigrostriatal lesions [34]
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254 These conditions share well-characterized pathogenic mechanismsincluding protein misfolding (ATTR-CM), 255 sarcomere dysfunction (HCM), 256 and intercellular adhesion instability (ARVC)which provide clear molecular targets for therapeutic intervention
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