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The main conclusion here, that excess SAM is catabolized to MTA and adenine, leading to methylation deficiencies, stands
Zlatar, M., Kokot, A., Vuleti, L., Masnec, S., Kralj, T., Peria, M., Barii, I., Radi, B., Milanovi, K., Drmic, D., Seiwerth, S., & Sikiric, P
Di Monaco, M
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Sevgiler et al., 2011), glutamic acid has been reported to chelate redox-active metals
10.1016/J.FERTNSTERT.2005.12.049 108 SandbergE
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