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This mechanism, while beneficial in controlled doses, can quickly become dangerous in cases of overdose, leading to a cascade of physiological responses that may require emergency intervention

None of these studies should be interpreted as recommending GHK-Cu for human use, treatment, or any clinical purpose

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A crucial observation was that DSIPs activating effect on oxidative phosphorylation was selective, primarily increasing V3 without affecting VDNP, and showing no evidence of uncoupling oxidative phosphorylation

Rodent physiology differs from human physiology in ways that are often relevant to drug development