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Similar content being viewed by others Introduction Despite the availability of new treatments over the last decade, a large proportion of people with type 2 diabetes (T2D) are not able to achieve their glycaemic goals [1, 2] because of clinical inertia (failure to begin or intensify treatment despite not reaching therapeutic goals) [3]
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doi:10.1038/s41569-023-00849-3 Wang W, Volkow ND, Berger NA, Davis PB, Kaelber DC, Xu R
This finding highlights not only the key role that mitochondrial metabolism has in modulating response and resistance to TS inhibitors, but the wider opportunity afforded to enhance TS inhibitor-mediated anti-cancer effects by targeting mitochondrial metabolism using metformin or other compounds with similar mitochondrial impact
Eur J Obstet Gynecol Reprod Biol 166:151155
The search strategy included combinations of the following keywords: GLP-1 receptor agonists, GLP-1 RA, gut microbiota, microbiome, short-chain fatty acids, bile acids, intestinal barrier, inflammation, and interindividual variability
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