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Inflammatory mediators released by perivascular cells such as microglia and astrocytes can cause BBB injury by increasing neuroinflammation

supplementing with GHK-Cu can have powerful regenerative properties of skin cells

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Mechanisms include: Restoration of endothelial nitric oxide synthase (eNOS) Upregulation of VEGF and FGF-2 (fibroblast growth factor 2) Reduction of oxidative stress and cytokine-induced apoptosis BPC-157 preserved endothelium-dependent vasodilation via upregulation of eNOS and suppression of vascular inflammation. Gwyer Findlay et al., Peptides 4