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Ferroptosis-inhibiting genes are enriched by GSH treatment We used RNA sequencing (RNA-seq) to identify differentially expressed genes (DEGs) between the treatment groups (Fig

Additionally, AK1C3 possesses 17-HSD activity and converts 4 -androstene-3,17-dione ( 4 -dione) to testosterone, as well as 3, 17-diol to androsterone 31

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Adjunctive treatment in clinical settings where enhancement of glutathione levels is considered beneficial for liver function and cellular protection

Intracellular GSH biosynthesis and intracellular levels can be upregulated as a protective mechanism