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The proposed mechanism involves maintaining synaptic vesicle integrity, competing for receptor sites, and potentially substituting for depleted neurotransmitters

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Also, several studies lacked full disclosure of variance measures or raw data, preventing the inclusion of potentially relevant findings

Second, we analyze how microbiota-derived metabolites (short-chain fatty acids, bile acids, tryptophan), gut hormones (glucagon-like peptide-1, ghrelin), and neurotransmitters (acetylcholine, vasoactive intestinal peptide, nitric oxide) multitarget the ENScollectively establishing the microbiota-ENS axis as the central hub for GI sensorimotor control