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a;Egecioglu et al., 2013c
The action of GLP-1 and GIP on the pancreas causes a reduction in glucagon secretion that results in diminished hepatic glucose production
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GLP-1 receptor (GLP-1R) signaling contributes to increased glucose-dependent insulin secretion, cell proliferation, islet size, portal glucose sensing, and postprandial lipid metabolism [3]
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DNA damage by azathymidine was observed both in experiments in cell cultures and motherchild pairs receiving this therapy 177
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