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GLP-1 receptor agonists work by mimicking incretin hormones that regulate insulin secretion, appetite signalling, and gastric function as described in clinical pharmacology literature [1]
Oral semaglutide offers a mechanism that supports appetite regulation and metabolic balance, making it suitable for patients who require more than lifestyle modification alone to achieve progress
Effects of GLP-1 and GIP receptor agonists include: Stimulating insulin secretion in response to food (glucose-dependent) Reducing inappropriate glucagon release, lowering hepatic glucose output (GLP-1 enhances the sensitivity of alpha cells to glucose , resulting in more glucose-appropriate glucagon secretion) When blood glucose is high, insulin secretion is stimulated and glucagon secretion is inhibited
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The current medical consensus, supported by the EMA's 2013 review and subsequent updates, is that whilst a small increased risk cannot be entirely excluded, the overall evidence does not establish a definitive causal link between GLP-1 agonist use and pancreatitis
55 Sebastian MonasorL.MllerS
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