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Critically, this redox adaptation is a key mechanism of resistance to radiotherapy and many chemotherapeutic agents, such as cisplatin and doxorubicin, which depend on generating oxidative damage to exert their cytotoxic effects [33]
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Differential expression analysis indicated a general upregulation of transcription in Gclc fl/fl MZB compared to Gclc fl/fl FoB (Fig
Is IGF1 LR3 better than HGH
PMID: 25038357 Badran AS, et al
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