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[3] In humans, defects in GSS are inherited in an autosomal recessive way and are the cause of severe metabolic acidosis, 5-oxoprolinuria, increased rate of haemolysis, and defective function of the central nervous system
Este hallazgo resulta coherente con el mecanismo de accin dual de este frmaco, que incluye la activacin del receptor GIP
Most cases were from the USA and France ( n = 6 each)
This means the cell never receives glutathione itself
aminopeptidase N receptor : 229E)
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