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This study showed that inhibiting HMGB1 nucleocytoplasmic translocation pharmacologically and deleting HMGB1 in TECs in mice were effective in preventing IRI-AKI, tubular ferroptosis, and inflammation compared to control groups [48]

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For the medications themselves, it was not very clear for any condition, though

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The trade-off: with-DAC trades closer-to-natural physiology for the convenience of weekly dosing