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Key Research Areas

Mitochondrial dysfunction leading to oxidative stress also induces FGF21 expression in preclinical disease models ( Enhanced secretory load, imbalances in redox homeostasis, or perturbations in protein homeostasis triggered by lysosomal or autophagosomal dysfunction may all cause ER stress and trigger the unfolded protein response (UPR)

[15] Animal studies in vivo An in vivo study in aging mice found that epitalon treatment significantly reduced the incidence of chromosomal aberrations, both for wild-type mice and for mice characterized by an accelerated aging phenotype, which is consistent with increases in telomere length

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These changes cannot occur without reducing the central cobalt ion from the + 3 oxidation state to + 2 or + 1 because cobalamins with a Co + 3 central ion are stable six-coordinate systems

Pre-medication with a bronchodilator may be recommended in such cases.