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These two heparin molecules are bound at the same site within the N domains of two HGFs and are surrounded by the same set of positively charged residues as described above
doi:10.1111/acel.12170
This issue has been attributed to the mode of action of these antagonists: those blocking CAP-, proton-, and heat-induced TRPV1 activation result in hyperthermia, whereas those sparing proton-induced activation do not lead to hyperthermia 33,35
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