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5), and by inference, neurotoxic damage.53 Forgione and colleagues had previously hypothesized that GPX1 deficiency directly induces an increase in vascular oxidative stress, with attendant endothelial dysfunction.54 Glutathione peroxidases, particularly GPx1 have also been implicated in the development and prevention of many common and complex diseases, including cancer and cardiovascular disease (Fig

Unfortunately, toxicity negatively affects rhizobium bacteria, root nodule formation, and the ultimate naturally plant-microbe-based N supply system [25]

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Direct Induction of CCK and GLP-1 Release from Murine Endocrine Cells by Intact Dietary Proteins. Molecular Nutrition & Food Research, vol