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Even though sulfur burps werent explicitly monitored in clinical trials, it can be inferred that they could potentially follow the same pattern
In mouse models of highfat dietinduced liver steatosis, it significantly inhibits hepatic NNMT activity, reduces NAM methylation, increases NAD+ and SAM levels, enhances mitochondrial fatty acid oxidation, reduces hepatic triglyceride and lipid accumulation, lowers lipotoxicity markers (e.g., malondialdehyde, transaminases), improves hepatocellular injury, and reverses steatosis
Cell Tissue Research
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For many patients, the disease can return monthsor even decadesafter initial treatment, this time in an entirely different organ
This connection between the gut & pancreatic islets may be responsible for ~50% of postprandial insulin release (Perley & Kipnis, J Clin Invest 46:1954-1962
