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This chronic increase in TBA-Rs compromises the biochemical integrity of the cell membranes of neurons in the prefrontal cortex, producing a lipid peroxidation process, altering plasticity, regenerative capacity, and dendritic architecture with shrinkage and decreased neuronal connectivity, leading to a decrease in BDNF and thus disturbing normal synaptic neurotransmission by oxidation of glutamate NMDA receptors, leading to attenuation of long-term potentials and synaptic neurotransmission 46
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