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The first study identified N -acetyl-aspartyl-glutamate (NAAG), which can be hydrolyzed to glutamate when needed by the oncogenic cells via glutamate carboxypeptidase II (GCP II) [41, 42], suggesting that GCPII is a viable target for cancer therapy, either alone or in combination with glutaminase inhibition
Metformin prevents methylglyoxal-induced apoptosis by suppressing oxidative stress in vitro and in vivo
Decreased interstitial SA--gal activity and lowered levels of senescence-associated proteins p53, p21, and p16
Epub ahead of print
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