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10.1081/CLT-100102388 Watson WA, Litovitz TL, Klein-Schwartz W, Rodgers GC Jr, Youniss J, Reid N, Rouse WG, Rembert RS, Borys D: 2003 annual report of the American Association of Poison Control Centers Toxic Exposure Surveillance System

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By activating the expression of SOD1, NRF2 helps maintain cellular redox balance and protects cells from oxidative injury [38]

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Across in vitro and preclinical models, GHK-Cu has been studied for its influence on gene expression programs related to matrix synthesis and remodeling, antioxidant defenses, and pathways associated with cellular senescence markers