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MOTS-c activates the AMPK pathway, which is the same pathway triggered by exercise and caloric restriction

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The study considered three hyperinsulinemia analogs of insulin, with PheB24Ser, PheB25Leu, and ValA3Leu mutations, and three more critical analogs after mutating the hinge residue TyrB26 to Ala, Glu, and Ser

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