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Abstract Background There are concerns that glucagon-like peptide-1 receptor agonists (GLP1s) increase the risk of acute pancreatitis
Although the impact of GLP-1RAs on pancreatic cancer risk remains controversial, emerging evidence suggests that chronic low-grade inflammation and proliferative changes may constitute key pathophysiological mechanisms underlying pancreatic cancer development in patients receiving GLP-1RA therapy [8]
GSS, glutathione synthetase
This can lead to: Intense, persistent nausea Projective or repetitive vomiting Severe abdominal cramping and bloating Significant dehydration due to the inability to keep fluids down Risk of Hypoglycemia While GLP-1 medications are generally known for having a low risk of causing low blood sugar (hypoglycemia) when used alone, taking excessive amounts changes the equation
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