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Glutaryl-CoA dehydrogenase (GCDH) deficiency is an inborn error of lysine and tryptophan metabolism that results in increased formation and excretion of glutaric acid (GA), 3-hydroxyglutaric acid (3-OH-GA), glutaconic acid and glutarylcarnitine [82]

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Adenosine modulates hypoxia-induced responses in rat PC12 cells via the A2A receptor J Physiol 1998

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The breakdown illustrates why evidence-based practitioners default to retinoids as the clinical standard while remaining appropriately open to GHK-Cu as a complementary option