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60 also increased phosphorylation of p38/MAPK and ERK/MAPK contributes to the decreased eNOS activity, 42 we hypothesized that eNOS -derived NO production may influence the healing of the gastric ulcer

The two independent pathways via glutathione reductases in cytosol and class III peroxidases in apoplast could maintain the balance of the GSH/GSSG cycle

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