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GHK-Cu has been studied for its role in collagen and elastin support, skin repair signaling, and matrix remodeling

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[13] In GPX1 (-/-) mice, oxidant formation is increased, endothelial NO synthase is deregulated, and adhesion of leukocytes to cultured endothelial cells is increased

This limited vascularity explains why tendon injuries heal so slowly through conventional rest alone