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Summary and perspectives This review highlights the pivotal role of amino acid metabolism in the pathogenesis of pulmonary fibrosis, with a focus on how dysregulated metabolic pathwaysparticularly those involving proline, arginine, glutamine, and tryptophandrive collagen deposition, fibroblast activation, and disease progression

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and reduced BMI (7%), waist circumference (3%), and abdominal fat (17%) (Aziz, Hussain, Mahwi, Ahmed, Rahman 2018)

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