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demonstrated that in PARP inhibitor-resistant TNBC, resulting from poly (ADP-ribose) polymerase 1 (PARP1) gene mutations, chemotherapy resistance can be reversed by inducing ferroptosis through the P53/SLC7A11 signaling pathway, via proteolysis-targeted chimaera (PROTAC) (65)

"Growth-modulating plasma tripeptide may function by facilitating copper uptake into cells"