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doi: 10.1016/j.chom.2019.05.003
2004;3(4):S1-S7
Longterm human studies are limited, so periodic lab work and physician followups remain essential.
N Engl J Med 354(15):15671577 Luka Z, Capdevila A, Mato JM, Wagner C (2006) A glycine N-methyltransferase knockout mouse model for humans with deficiency of this enzyme
Parallel animal models in the same study revealed that glutathione supplementation increased mitochondrial DNA by 53% and improved intermuscular pH, effectively preventing the acidic environment that leads to rapid muscle burning and exhaustion
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