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With continued proliferation of nevus cells, they extend from the dermalepidermal junction into the dermis (forming a compound nevus)
10.1007/s00223-004-0147-4
& Boyle, C
In animal models, DSIP administration has been associated with reduced markers of oxidative damage and improved mitochondrial function, supporting its use in studies of neuronal resilience and metabolic stability [6]
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